4. A sportsman is doing a 10 km marathon for 1.5 hours. What changes in lipid metabolism in adipose tissue and muscles are triggered by long-term physical activity? To answer the question: a) name the hormonc, controlling the lipid metabolism in adipocytes and write down the scheme of the process; b) explain, how some of the products of lipid mobilization, transported from adipocytes to the bloodstream, would be utilized by skeletal muscles and liver in these conditions; c) calculate the ATP vicld in the result of palmitolcic acid oxidation.
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- 4. A sportsman is doing a 10 km marathon for 1.5 hours. What changes in lipid metabolism in adipose tissue and muscles are triggered by long-term physical activity? To answer the question: a) name the hormone, controlling the lipid metabolism in adipocytes and write down the scheme of the process; b) explain, how some of the products of lipid mobilization, transported from adipocytes to the bloodstream, would be utilized by skeletal muscles and liver in these conditions; c) calculate the ATP yicld in the result of palmitolcic acid oxidation.3. After 12 days of starvation, a man reduced his weight by 4 kg. The doctor prescribed him biochemical blood test to analyze the level of lipids in serum blood. The results showed an elevated level of free fatty acids and triacylglycerols. Explain the results of the biochemical blood test. For that: a) name the main hormone which manages metabolism during starvation; b) draw the scheme of this hormone action on the adipose tissue; c) name the possible pathways of fatty acids usage in the liver during starvation and write down the proper schemes.8. Ketone bodies were found in the urine of the patient with diabetes mellitus. Explain the sequence of metabolic changes resulting in ketonuria. For that answer the question and do the following tasks: a) what are the ranges of ketone bodics in blood of the healthy pcople and the patients with diabetes mellitus? b) explain the main changes in hormonal regulation of patient with diabetes mellitus; c) explain why the level of free fatty acids in the blood is clevated; d) name the pathways of lipid metabolism which becomes more active in patients with diabetes mellitus; e) draw the scheme of ketone body synthesis and oxidation.
- 3. A patient has got excess carbohydrate meal for the years and gain the weight. To explain this: a) draw the schemes of TAG synthesis in the liver; b) describe the transport of TAG from the liver to adipose tissue; c) describe the functions of insulin in the conversion of glucose to TAG in the liver and adipose tissue. Glucose containing Catoms was added to isolated hepatocytes inanexperiment. Ifthe glucose was added in excess, the rate of triacylglyccrol synthesis increased.1. Discuss why a saturated fatty acid like lauric acid has a good anti-oxidant property. 2. Why are the essential fatty acid associated with low incidence of heart disease? Cite some clinical signs of essential fatty acid deficiency. 3. Explain how aspirin can block the synthesis of prostaglandins?8. In patients with diabetes mellitus type I, the biochemical disorders result from changes in fuel metabolism. One of these signs is acidosis. Explain why such patients have a deviation of blood pH from the norm? For this: d) specify the hormone that accelerates this precursor formation and provide appropriate charts, starting from the hormone binding to adipocyte and concluding with precursor formation, give an explanation to the charts.
- 1a. Type II Diabetes involves the development of insulin resistance (i.e. a dampening of the metabolic response to insulin). Describe the metabolic effects of such resistance. 1b. What would the likely metabolic effects be of a person being hypersensitive to insulin?8. In patients with diabetes mellitus type I, the biochemical disorders result from changes in fucl metabolism. One of these signs is acidosis. Explain why such patients have a deviation of blood pH from the norm? For this me the Ocuics, name d) specify the hormone that accelerates this preeursor formation and provide appropriate charts, starting from the hormone binding to adipocyte and concluding with precursor formation, give an explanation to the charts.The hormones insulin and glucagon play an important role in the regulation of plasma glucose.a) Discuss the antagonistic actions of the hormones insulin and glucagon in regulating blood glucose levels within a narrow physiological range.
- A person is suspected with diabetes mellitus. He checks his blood glucose level by gluceose oxidase method. After the experiment, OD of his blood sample was obtained as 0.08. Based on the glucose standard curve seen in the virtual lab, calculate his blood glucose level. Is he had diabetes mellitus? (Note: Normal blood glucose level in our body is 70-110mg/dl). Find out the role and chemical reactions of the following reagents in the blood glucose level determination by glucose oxidase method. a) Sodium sulphate – Zinc sulphate solution b ) Glucose oxidase reagent c) Peroxidase A person did estimation of blood glucose level by glucose oxidase method by referring the virtual lab procedure. But he forgot to add ortho - toluidine reagent during glucose oxidase reagent preparation. a) What is the observation of the result?…(27) An 80-year old man witha 20-year old history of ashthma comes to the physician because of a 24 hour history of a running nose , sore throat , cough,muscle aches, and fatigue. His temperature is 39.5 degrees celcius (103.1F). Zanamivir is prescribed because it inhibits which of the following enzymes? (A) Aldolase (B) Kinase (C) N-acetyl glucosaminidase (D) Neuraminidase (E) TransglycosylaseCompare and contrast the following items related to lipid metabolism. Cite their main similarities and/or differences in not more than 5 sentences each: 1. lysophosphatidylcholine vs phosphatidylethanolamine 2. trimyristin vs triolein 3. ACP vs carnitine-acyl transferase 4. dehydrogenase enzyme vs dehydratase enzyme in the context of lipid metabolism) 5. steroid hormones vs prostaglandins (in terms of their biosynthetic pathways) 6. fatty acid synthase complex vs pyruvate dehydrogenase complex