Two genetic crosses have been proposed between male and female mice with varying hair length. These traits are described in the table below. For each genetic cross, complete the sentence that describes the predicted outcome of the cross. Use a separate sheet of paper to construct a Punnett square for each cross to accurately make your predictions. Allele Dominant or recessive? Trait description dominant short hair b recessive long hair 1. Baby mice produced from the cross between male BB and female bb parents will include Choose genotypes and Choose- phenotypes. 2. Baby mice produced from the cross between male Bb and female Bb parents will include Choose - genotypes and Choose - phenotypes.
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- I. ACTIVITIES AND EXERCISES-Let's Try Thesef () Solve for the following completely by following the step by step process in solving for Punnett Squares. Place your answers on the corresponding blanks. White skin tone is incompletely dominant with Dark Skin tone. A heterozygous skin tone allele will form Tan colored skin. What is/are the possible phenotype/s of the children from a white skin toned man and a tan skin toned woman? Traits: White Phenotype/s: Dark => Tan == Test Cross: Punnett Square 14) 2) 3) 8) 9) 5) 10) 12) 7) 11) Teacher: 13)Trivla Game Show _Make Your Own Tri ngston.schoology.com/common-assessment-delivery/start/4789189591?action=onresume&submissionld=463322566 Dillon WF g Aa v Done In guinea pigs, black hair (B) is dominant to white hair (b) and rough hair (R) is dominant to smooth hair (r). What are all the possible genotypes of a guinea pig that has black, rough hair? (Select all that apply.) O BBRR BBRr BBrr BBRR BbRr O bbRR O bbRr O bbrr O Black O White O Rough OSmooth O Rough O SmoothHi, I'm having trouble with my study guide for my upcoming genetics exam. If someone could please help with work shown and an explanation it would help so much! Thank you!! 2a. The pedigree below represents inheritance of rare condition. What pattern of inheritance is most consistent with the data? Assign alleles to all individuals to support your answer. If an allele is unknown, assign it a ? symbol. NOTE: Individuals whose phenotype or genotype cannot be determined are assumed to be unaffected and homozygous, unless otherwise indicated. 2b. In addition to the alleles you’ve indicated, describe 2 overall features of the pedigree that make it consistent with your chosen form of inheritance. 2c. Based on your mode of inheritance, what is the probability that the child of couple IV-4 x IV-5 will be affected? Show your work. attached is the pedigree
- Switch Background P Immersive Reader 100% Page Width d. What percent of the offspring will be carriers of the white eye trait? 2. Using the same information as for question #1, cross a heterozygous red-eyed female with a red-eyed male. a. What are the genotypes of each parent? ic b. What fraction of the children will have red eyes? c. What fraction of the children will have white eyes? Pra d. What fraction of the female children will carry the white eyed trait?Drosohpila Punnet Square of Crosses. I need results of F1 & F2 generation using Punnett Squares for: Make Punnet Squares of the following crosses •Drosophila Female wildtype cross Male White-eye •Drosophila Male wildtype cross Female White-eye •Drosophila Female Wild Type cross Male Scarlet Eye •Drosophila Male Wild Type cross Female Scarlet Eye Also, Which allele is heterozygous and which is homozygous, & which is dominant and which is recessive?Bb Take Test: Mod 9 Lab - Genetic X + ← ] C G QUESTION 3 In the pedigree below, all shaded individuals express the gene in question. For example, Arlene "has" the trait, she displays the phenotype in question. For example, if we were following the inheritance pattern of a widow's peak, Arlene has a widow's peak (that is NOT the trait here, just an example). Unshaded individuals (blank circles and squares) do not manifest the trait in question, but their specific genotype is unknown - they could be heterozygous, homozygous dominant, or homozygous recessive. What is the mechanism of inheritance of this trait? Sandra Daniel recessive dominant Tom 990 Alan QUESTION 4 George Sam Tina Christopher Arlene Wilma Ann O Carla R Update Michael In the pedigree below, all shaded individuals express the gene in question. For example, Arlene "has" the trait, she displays the phenotype in question. For example, if we were following the inheritance pattern of a widow's peak, Arlen a widow's peak (that…
- DATA | The following pedigree illustrates the inheritance of ringed hair, a condition in which 28. ANALYSIS each hair is differentiated into light and dark zones. What mode or modes of inheritance are possible for the ringed-hair trait in this family? 2 II 2 II 3 4 5 IV I P 2 Pierce, Genetics: A Conceptual Approach, 7e © 2020 W. H. Freeman and Company 2.ng -Courses iblic/activ 003004/a s sment al V T-Rex Game. un in to your acc. L 1.3.4 Quiz: Predicting Genetic Outcomes Question 1 of 10 Mendel used over 28,000 pea plants in his experiment. How does this large sample size make his results more reliable? O A. He was not sure which of the plants were female and which were male. B. Most of the plants were unable to reproduce, so he needed many of them. C. He used more plants so the experiment wouldn't take as long. D. It made his actual results approach the results predicted by probability. SUBMIT E PREVIOUSA couple was referred for genetic counseling because they wanted to know the chances of having a child with dwarfism. Both the man and the woman had achondroplasia (MIM 100800), the most common form of short-limbed dwarfism. The couple knew that this condition is inherited as an autosomal dominant trait, but they were unsure what kind of physical manifestations a child would have if it inherited both mutant alleles. They were each heterozygous for the FGFR3 (MIM 134934) allele that causes achondroplasia. Normally, the protein encoded by this gene interacts with growth factors outside the cell and receives signals that control growth and development. In achrodroplasia, a mutation alters the activity of the receptor, resulting in a characteristic form of dwarfism. Because both the normal and mutant forms of the FGFR3 protein act before birth, no treatment for achrondroplasia is available. The parents each carry one normal allele and one mutant allele of FGRF3, and they wanted information on their chances of having a homozygous child. The counsellor briefly reviewed the phenotypic features of individuals with achondroplasia. These include facial features (large head with prominent forehead; small, flat nasal bridge; and prominent jaw), very short stature, and shortening of the arms and legs. Physical examination and skeletal X-ray films are used to diagnose this condition. Final adult height is approximately 4 feet. Because achondroplasia is an autosomal dominant condition, a heterozygote has a 1-in-2, or 50%, chance of passing this trait to his or her offspring. However, about 75% of those with achondroplasia have parents of average size who do not carry the mutant allele. In these cases, achondroplasia is due to a new mutation. In the couple being counseled, each individual is heterozygous, and they are at risk for having a homozygous child with two copies of the mutated gene. Infants with homozygous achondroplasia are either stillborn or die shortly after birth. The counselor recommended prenatal diagnosis via ultrasounds at various stages of development. In addition, a DNA test is available to detect the homozygous condition prenatally. What if the couple wanted prenatal testing so that a normal fetus could be aborted?
- A couple was referred for genetic counseling because they wanted to know the chances of having a child with dwarfism. Both the man and the woman had achondroplasia (MIM 100800), the most common form of short-limbed dwarfism. The couple knew that this condition is inherited as an autosomal dominant trait, but they were unsure what kind of physical manifestations a child would have if it inherited both mutant alleles. They were each heterozygous for the FGFR3 (MIM 134934) allele that causes achondroplasia. Normally, the protein encoded by this gene interacts with growth factors outside the cell and receives signals that control growth and development. In achrodroplasia, a mutation alters the activity of the receptor, resulting in a characteristic form of dwarfism. Because both the normal and mutant forms of the FGFR3 protein act before birth, no treatment for achrondroplasia is available. The parents each carry one normal allele and one mutant allele of FGRF3, and they wanted information on their chances of having a homozygous child. The counsellor briefly reviewed the phenotypic features of individuals with achondroplasia. These include facial features (large head with prominent forehead; small, flat nasal bridge; and prominent jaw), very short stature, and shortening of the arms and legs. Physical examination and skeletal X-ray films are used to diagnose this condition. Final adult height is approximately 4 feet. Because achondroplasia is an autosomal dominant condition, a heterozygote has a 1-in-2, or 50%, chance of passing this trait to his or her offspring. However, about 75% of those with achondroplasia have parents of average size who do not carry the mutant allele. In these cases, achondroplasia is due to a new mutation. In the couple being counseled, each individual is heterozygous, and they are at risk for having a homozygous child with two copies of the mutated gene. Infants with homozygous achondroplasia are either stillborn or die shortly after birth. The counselor recommended prenatal diagnosis via ultrasounds at various stages of development. In addition, a DNA test is available to detect the homozygous condition prenatally. What is the chance that this couple will have a child with two copies of the dominant mutant gene? What is the chance that the child will have normal height?A couple was referred for genetic counseling because they wanted to know the chances of having a child with dwarfism. Both the man and the woman had achondroplasia (MIM 100800), the most common form of short-limbed dwarfism. The couple knew that this condition is inherited as an autosomal dominant trait, but they were unsure what kind of physical manifestations a child would have if it inherited both mutant alleles. They were each heterozygous for the FGFR3 (MIM 134934) allele that causes achondroplasia. Normally, the protein encoded by this gene interacts with growth factors outside the cell and receives signals that control growth and development. In achrodroplasia, a mutation alters the activity of the receptor, resulting in a characteristic form of dwarfism. Because both the normal and mutant forms of the FGFR3 protein act before birth, no treatment for achrondroplasia is available. The parents each carry one normal allele and one mutant allele of FGRF3, and they wanted information on their chances of having a homozygous child. The counsellor briefly reviewed the phenotypic features of individuals with achondroplasia. These include facial features (large head with prominent forehead; small, flat nasal bridge; and prominent jaw), very short stature, and shortening of the arms and legs. Physical examination and skeletal X-ray films are used to diagnose this condition. Final adult height is approximately 4 feet. Because achondroplasia is an autosomal dominant condition, a heterozygote has a 1-in-2, or 50%, chance of passing this trait to his or her offspring. However, about 75% of those with achondroplasia have parents of average size who do not carry the mutant allele. In these cases, achondroplasia is due to a new mutation. In the couple being counseled, each individual is heterozygous, and they are at risk for having a homozygous child with two copies of the mutated gene. Infants with homozygous achondroplasia are either stillborn or die shortly after birth. The counselor recommended prenatal diagnosis via ultrasounds at various stages of development. In addition, a DNA test is available to detect the homozygous condition prenatally. Should the parents be concerned about the heterozygous condition as well as the homozygous mutant condition?GsnKivd010j2gIRWLIZOMZZ-VibKYvBbo61ylATAQ/viewform RECOMBINATION". For numbers 7-35, reler to the given data below. Glven the following testcross data for com In whlch the genes for fine stripe (f), bronze gleurone (bz) and knotted leaf (Kn) are involved: + = wild type f fine stripe +=wild type bz = bronze gleurone +=wild type Kn knotted leaf %3D Genotype Ko f Number 451 Ko 134 97 436 bz bz bz Ko 18 119 f 24 Kn f bz 86 Total: Your answer 7-8. What would be the recombination frequency or the frequency of the recombinant type between +/Kn and +/f genes? Oa. 16% O b. 16 map units + + + + +