During high-intensity exercise, why does arterial Pco2 decrease beyond its usual set point? Arterial Po2 is reduced during high intensity exercise, which stimulates chemoreceptors to increase ventilation beyond current CO2 production. O During high intensity exercise, the blood becomes more alkaline, which leads to more CO₂ being converted to H* and bicarbonate. Anaerobic metabolism does not produce CO2. O Acidic by-products of anaerobic metabolism decrease plasma pH, which stimulates central chemoreceptors to increase ventilation beyond current CO₂ production.

During high-intensity exercise, why does arterial Pco2 decrease beyond its usual set point? Arterial Po2 is reduced during high intensity exercise, which stimulates chemoreceptors to increase ventilation beyond current CO2 production. O During high intensity exercise, the blood becomes more alkaline, which leads to more CO₂ being converted to H* and bicarbonate. Anaerobic metabolism does not produce CO2. O Acidic by-products of anaerobic metabolism decrease plasma pH, which stimulates central chemoreceptors to increase ventilation beyond current CO₂ production.

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter13: The Respiratory System

Section: Chapter Questions

Problem 3TAHL

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Which of the following statements regarding control of respiration is TRUE? At high altitude, a decrease in PC02 of the blood stimulates an increase in ventilation. An increase in the HCO3- concentration in blood stimulates ventilation. A slight decrease in arterial PO2 is a stronger stimulus for increased ventilation than is a comparable decrease in arterial PCO2. The most important signal for regulating ventilation is the H+ concentration of arterial blood. Increased concentrations of lactic acid stimulate ventilation primarily by acting on peripheral chemoreceptors.
You notice a significant increase in your rate of breathing following a hard set of bench press. This is likely. Hyperventilation to compensate for the acute increase in lactic acid Hypoventilation to compensate for the acute increase in lactic acid O Hyperventilation to increase the levels of blood CO2. Hypoventilation to compensate for the muscular production of biocarbonate O Hyperventilation to compensate for the muscular production of bicarbonate
After light exercise, the oxygen consumed in recovery is approximately equal to the oxygen deficit, which is the amount of additional oxygen that would have been consumed had oxygen consumption reached steady state immediately. How is the oxygen consumed in recovery used?
The physiological mechanisms which have caused respiratory rate to change during exercise. Before exercise Results are: RR 14 bpm, SpO2 98%, BP 130/70 mmHg (Mean arterial pressure 90 mmHg), HR 74 bpm. No cardiovascular or respiratory diseases. After exercise respiratory rate is now 20 bpm, her SpO2 is 100% on room air, BP is 140/80 mmHg (Mean arterial pressure 100 mmHg), and HR is 90 bpm. Breathing deeply and using accessory muscles.
during exercise what organ is most responsible for the increased production of carbon dioxide? Brain, skeletal muscles, or heart.
At rest the typical partial pressure of CO2 in arterial blood is around 40mmHg, and the ventilation rate is around 15 breathes per minute. During vigorous exercise you would expect the partial pressure of CO2 in the arterial blood to which would cause the ventilation rate to to compensate and restore homeostasis Increase; decrease Decrease; decrease Increase; increase Decrease; increase
Anaerobic respiration releases ATP faster than aerobic respiration. True or False?
Altitude effects on exercise are caused mainly by increased CO2 production due to the physiological response to low pressure the increased influence of nitrogen gas at higher altitudes lowered 02 partial pressure the direct effect of low pressure on breathing
Oxygen uptake (Fick Equation) is determined by two variables. What are they and what do they represent
Messner and Habeler's 1978 ascent of Mount Everest without oxygen-breathing apparatus is often described as one of the most remarkable physical accomplishments achieved by humans. Table 1 shows respiratory gas and arterial pH values measured in a resting mountaineer at sea level, Everest base camp and Everest summit. Use the differences in these values to explain how breathing regulation changes when at rest at the 3 altitudes indicated. Calculations are not required but you may wish to consider the role of central and peripheral chemoreceptors and their relationship to paCO,, minute ventilation and alveolar pO, in your response. Table 1: Respiratory gas composition and arterial pH measured in a resting mountaineer at sea-level, Everest base-camp and Everest summit. Arterial Barometric Alveolar Inspíred p02 (FIO2) pO2 (PAO2) pCO2 (расо2) Altitude Pressure Arterial (m) pH mmHg mmHg mmHg mmHg Sea Level 760 150 106 36 7.4 Everest Base 5,400 404 75 51 20.4 7.6 Camp Everest 8,848 252 43 34…
The resting O2 saturation for the subject was 98%, and it was still 98% during exercise. What information does this give you about the signal that triggers a change in ventilation rate during exercise?
Define the Bohr effect and describe how it explains maximal oxygen delivery to tissues in greatest need.